The Effect of Secondhand Smoke Exposure on Bronchus Airway Inflammation in COPD

Dear Editor

Secondhand smoke exposure contributes to the development of Chronic Obstructive Pulmonary Disease (COPD) with occupational exposure and pollution from indoor fires being significant causes in some countries, and there is convincing evidence that bronchus inflammation may alter COPD individual susceptibility [1]. The primary cause of COPD is tobacco smoke, and the secondary is secondhand smoking exposure. The primary risk factor for COPD of those who smoke, about 20% will get COPD, and of those who are lifelong smokers, about half will get COPD [2]. According to estimates by the World Health Organization, the prevalence of the global population is around 5%. morbidity and mortality are currently [3]. Chronic inflammation is both a causal factor due to secondhand smoke exposure and a consequence of most chronic diseases of asthma and COPD.

However, this chronic inflammation process can persist over an extended period of weeks to months and even years [4]. Secondhand smoke exposure is a major cause of human Chronic Obstructive Pulmonary Disease (COPD). COPD has pathophysiological features of diseases [5]. Physiological COPD is individuals who cause poor lung development or growth for a variety of reasons during pregnancy, neonatal, infancy, or childhood [6]. Exercise is a good way to increase the rate of metabolism. Exercise improves heart rate and increases the rate of metabolism and burning of heat. For people who have many years of smoking, it is important to start exercising. Make sure to drink plenty of water because nicotine is soluble in water, so drinking water helps to excrete the substance through the urine [7]. Vitamin A is also helpful in removing nicotine from the body because it also has the effect of speeding up the metabolism [8].

Physiological COPD is an airway responsiveness. Physiological COPD is controlled by inherited genes. However, pathological COPD is an environmental factor. Secondhand smoke exposure is the risk of pathological COPD [9]. Therefore, chronic inflammation plays a critical role in COPD disease processes.

Acknowledgment

None.

Conflicts of Interest

None.

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